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Characterising COVID-19 as a Viral Clotting Fever: A Mixed Methods Scoping Review | medRxiv
Issue: Cell
Kinetics of antibody responses dictate COVID-19 outcome | medRxiv
Hydrogen sulfide dysregulates the immune response by suppressing central carbon metabolism to promote tuberculosis | PNAS
Transcriptomic similarities and differences in host response between SARS-CoV-2 and other viral infections - ScienceDirect
Issue: Cell
Characterising COVID-19 as a Viral Clotting Fever: A Mixed Methods Scoping Review | medRxiv
Issue: Cell
High titers and low fucosylation of early human anti–SARS-CoV-2 IgG promote inflammation by alveolar macrophages
Hydrogen sulfide dysregulates the immune response by suppressing central carbon metabolism to promote tuberculosis | PNAS
Transcriptomic similarities and differences in host response between SARS-CoV-2 and other viral infections - ScienceDirect
High titers and low fucosylation of early human anti–SARS-CoV-2 IgG promote inflammation by alveolar macrophages
Characterising COVID-19 as a Viral Clotting Fever: A Mixed Methods Scoping Review | medRxiv
High titers and low fucosylation of early human anti–SARS-CoV-2 IgG promote inflammation by alveolar macrophages
Transcriptomic similarities and differences in host response between SARS-CoV-2 and other viral infections - ScienceDirect
Hydrogen sulfide dysregulates the immune response by suppressing central carbon metabolism to promote tuberculosis | PNAS
Kinetics of antibody responses dictate COVID-19 outcome | medRxiv
Characterising COVID-19 as a Viral Clotting Fever: A Mixed Methods Scoping Review | medRxiv
Bacille Calmette-Guérin induces NOD2-dependent nonspecific protection from reinfection via epigenetic reprogramming of monocytes | PNAS
Issue: Cell
Transcriptomic similarities and differences in host response between SARS-CoV-2 and other viral infections - ScienceDirect
High titers and low fucosylation of early human anti–SARS-CoV-2 IgG promote inflammation by alveolar macrophages
High titers and low fucosylation of early human anti–SARS-CoV-2 IgG promote inflammation by alveolar macrophages
High titers and low fucosylation of early human anti–SARS-CoV-2 IgG promote inflammation by alveolar macrophages
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